Overweight, hungry, diabetic, and fat-free

Let me tell you about my low-fat experience from 20 years ago.

At the time, I was living in Cleveland, Ohio, and served on the faculty at a large metropolitan university-affiliated hospital, supervising fellows-in-training and developing high-tech cath lab procedures like directional athererectomy and excimer laser coronary angioplasty. (Yes, another life.)

I was concerned about personal heart disease risk, though I knew next to nothing about lipids and coronary risk prediction outside of the little I learned in training and what the drug industry promoted.

I heard Dr. Dean Ornish talk while attending the American College of Cardiology meetings in Atlanta. Dr. Ornish spoke persuasively about the dangers of fat in the diet and how he "reversed" coronary disease using a low-fat, no added oils, no meat, vegetarian diet that included plenty of whole grains. So I thought I'd give it a try.

I eliminated all oils; I removed all meat, eggs, and fish from my diet. I shunned all nuts. I ate only low-fat products like low-fat yogurt and cottage cheese; and focused on vegetables, fruit, and whole grains. Beans and brown or wild rice were a frequent staple. I loved oatmeal cookies--low-fat, of course!

After one year of this low-fat program, I had gained a total of 31 lbs, going from 155 lbs to 186 lbs. I reassessed some basic labs:

HDL 28 mg/dl
Triglycerides 336 mg/dl
Blood sugar 151 mg/dl (fasting)


I became a diabetic. All through this time, I was also jogging. I ran on the beautiful paths along the Chagrin River in suburban Cleveland for miles north and south. I ran 5 miles per day most days of the week.

It was diabetes that hit me alongside the head: I was eating low-fat meticulously, exercising more than 90% of the population, yet I got fat and diabetic!

I have since changed course in diet. Last time I checked, my lipid values on NO statin agent:

HDL 67 mg/dl
Triglycerides 57 mg/dl
Blood sugar 91 mg/dl

That was my lesson that fat restriction is a destructive, misguided notion. The data since then have confirmed that restricting total fat is unnecessary, even undesirable, when fat calories are replaced by carbohydrate calories.

This is your brain on wheat

Here's just a smattering of the studies performed over the past 30 years on the psychological effects of wheat consumption.

Oddly, this never makes the popular press. But wheat underlies schizophrenia, bipolar illness, behavioral outbursts in autism, Huntington's disease, and attention deficit hyperactivity disorder (ADHD).

The relationship is especially compelling with schizophrenia:

Opioid peptides derived from food proteins: The exorphins.
Zioudrou C et al 1979
"Wheat gluten has been implicated by Dohan and his colleagues in the etiology of schizophrenia and supporting evidence has been provided by others. Our experiments provide a plausible biochemical mechanism for such a role, in the demonstration of the conversion of gluten into peptides with potential central nerovus system actions."


Wheat gluten as a pathogenic factor in schizophrenia
Singh MM et al 1976
"Schizophrenics maintained on a cereal grain-free and milk-free diet and receiving optimal treatment with neuropleptics showed an interruption or reversal of their therapeutic progress during a period of "blind" wheat gluten challenge. The exacerbation of the disease process was not due to variations in neuroleptic doses. After termination of the gluten challenge, the course of improvement was reinstated. The observed effects seemed to be due to a primary schizophrenia-promoting effect of wheat gluten."


Demonstration of high opioid-like activity in isolated peptides from wheat gluten hydrolysates
Huebner FR et al 1984


Is schizophrenia rare if grain is rare?
Dohan FC et al 1984
"Epidemiologic studies demonstrated a strong, dose-dependent relationship between grain intake and the occurrence of schizophrenia."

Small LDL: Perfect index of carbohydrate intake

Measuring the number of small LDL particles is the best index of carbohydrate intake I know of, better than even blood sugar and triglycerides.

In other words, increase carbohydrate intake and small LDL particles increase. Decrease carbohydrates and small LDL particles decrease.

Why?

Carbohydrates increase small LDL via a multistep process:

First step: Increased fatty acid and apoprotein B production in the liver, which leads to increased VLDL production. (Apoprotein B is the principal protein of VLDL and LDL)

Second step: Greater VLDL availability causes triglyceride-rich VLDL to interact with other particles, namely LDL and HDL, enriching them in triglycerides (via the action of cholesteryl-ester transfer protein, or CETP). Much VLDL is converted to LDL.

Third step: Triglyceride-rich LDL is "remodeled" by enzymes like hepatic lipase, which create small LDL.


Carbohydrates, especially if they contain fructose, also prolong the period of time that triglyceride-rich VLDL particles persist in the blood, allowing more time for VLDL to interact with LDL.

Many people are confused by this. "You mean to tell me that reducing carbohydrates reduces LDL cholesterol?" Yes, absolutely. While the world talks about cutting saturated fats and taking statin drugs, cutting carbohydrates, especially wheat (the most offensive of all), cornstarch, and sugars, is the real key to dropping LDL.

However, the effect will not be fully evident if you just look at the crude conventional calculated (Friedewald) LDL cholesterol. This is because restricting carbohydrates not only reduces small LDL, it also increases LDL particle size. This make the calculated Friedewald go up, or it blunts its decrease. Conventional calculated LDL will therefore either underestimate or even conceal the real LDL-reducing effect.

The reduction in LDL is readily apparent if you look at the superior measures, LDL particle number (by NMR) or apoprotein B. Dramatic reductions will be apparent with a reduction in carbohydrates.

Small LDL therefore serves as a sensitive index of carbohydrate intake, one that responds literally within hours of a change in food choices. Anyone following the crude Friedewald calculated LDL will likely not see this. This includes the thousands of clinical studies that rely on this unreliable measure and come to the conclusion that a low-fat diet reduces LDL cholesterol.

Fat "conditioning"

Here's a great study from the prolific laboratory of Dr. Jeff Volek from the University of Connecticut. (Full text here.)


http://jn.nutrition.org/cgi/content/full/134/4/880

Video Teleconference with Dr. William Davis


Dr. Davis is available for personal
one-on-one video teleconferencing

to discuss your heart health issues.


You can obtain Dr. Davis' expertise on issues important to your health, including:

Lipoprotein assessment

Heart scans and coronary calcium scores

Diet and nutrition

Weight loss

Vitamin D supplementation for optimal health

Proper use of omega-3 fatty acids/fish oil



Each personalized session is 30 minutes long and by appointment only. To arrange for a Video Teleconference, go to our Contact Page and specify Video Teleconference in your e-mail. We will contact you as soon as possible on how to arrange the teleconference.


The cost for each 30-minute session is $375, payable in advance. 30-minute follow-up sessions are $275.

(Track Your Plaque Members: Our Member cost is $300 for a 30-minute session; 30-minute follow-up sessions are $200.)

After the completion of your Video Teleconference session, a summary of the important issues discussed will be sent to you.

The Video Teleconference is not meant to replace the opinion of your doctor, nor diagnose or treat any condition. It is simply meant to provide additional discussion about your health issues that should be discussed further with your healthcare provider. Prescriptions cannot be provided.

Note: For an optimal experience, you will need a computer equipped with a microphone and video camera. (Video camera is optional; you will be able to see Dr. Davis, but he will not be able to see you if you lack a camera.)

We use Skype for video teleconferencing. If you do not have Skype or are unfamiliar with this service, our staff will walk you through the few steps required.

Track Your Plaque challenges

Of all the various factors we correct in the Track Your Plaque program in the name of achieving reversal of coronary plaque, there are two factors that are proving to be our greatest challenges:

1) Genetic small LDL

2) Lipoprotein(a)

More and more people are enjoying at least marked slowing, if not zero change or reduction, in heart scan scores following the Track Your Plaque program. We achieve this by correcting a number of factors. Some factors, like vitamin D deficiency, are easily corrected to perfection--supplement sufficient vitamin D to achieve a blood level of 25-hydroxy vitamin D of 60-70 ng/ml. Correcting standard lipid values--LDL cholesterol, HDL cholesterol, and triglycerides--child's play, even to our strict targets of 60-60-60.

However, what I call "genetic small LDL" and a subset of lipoprotein(a) are proving to be the most resistant of all.

Let's first consider genetic small LDL. Small LDL is generally the pattern of the carbohydrate-ingesting, overweight person. It has exploded in severity over the past decade due to overconsumption of carbohydrates due to the ridiculous low-fat notion. Reduce or eliminate carbohydrates, especially wheat, which permits weight loss, and small LDL drops like a stone. But there is a unique subset of people who express the small LDL pattern who start at or near ideal weight. Take Chad, for instance. At 6' 2" and 152 lbs and BMI of 19.6, there's no way excess weight could be triggering his small LDL. Yet he starts with 100% small LDL particles. All efforts to reduce small LDL, such as wheat, cornstarch, and sugar elimination; niacin; vitamin D normalization; thyroid normalization; and several supplements that yield variable effects, such as phosphatidylcholine, all leave Chad with more than 90% small LDL.

Lipoprotein(a) is a bit different. Over the past 5 years, our choices in ways to reduce Lp(a) expression have improved dramatically. Beyond niacin, we now have high-dose EPA + DHA, thyroid normalization that includes use of T3, and hormonal manipulation. In the Track Your Plaque experience, approximately 70% of people with Lp(a) respond with a reduction in Lp(a). (In fact, the 4 out of the 5 record holders for reduction of heart scan scores have Lp(a) that was successfully treated.) But about 30% of people with Lp(a) prove resistant to all these treatments--they begin with a Lp(a) of, say, 260 nmol/L and, despite niacin, high-dose EPA + DHA, and various hormones, stay at 260 nmol/L. It can be frustrating and frightening.

So these are the two true problem areas for the Track Your Plaque program, genetic small LDL and a subset of Lp(a).

We are actively searching for better options for these two problem areas. Given the collective exploration and wisdom that develops from such collaborative efforts as the Track Your Plaque Forum, I am optimistic that we will have better answers for these two stumbling blocks to plaque reversal in the future.

I'll supply the tar if you supply the feathers

The results of the latest Heart Scan Blog poll are in.


DIRECT-TO-CONSUMER PHARMACEUTICAL ADVERTISING HAS:

Increased public awareness of medical conditions and their treatment
19 (11%)

Has had little overall effect on health and healthcare
29 (18%)

Needlessly increased healthcare costs
81 (50%)

Further empowered the revenue-obsessed pharmaceutical industry
130 (81%)


Clearly, there's a lot of negative sentiment against direct-to-consumer (DTC) drug advertising.

It looks as if a small minority believe that good has come from DTC advertising, judging by the meager 11% who voted for increased awareness. In fact, the poll results are heavily weighed towards the negative: 50% voted for "needlessly increased healthcare costs," while an astounding 81% voted for "empowered the revenue-obsessed pharmaceutical industry."

It is, indeed, an odd situation: Pharmaceutical agents available only by prescription being hyped directly to the consumer.

Personally, I would vote for choices 1,3, and 4. While awareness has increased, it has come with a hefty price, not all of it well spent. I believe the pharmaceutical industry still adheres to the rule that, for every $1 spent on advertising, $4 is made in revenue. They are, in effect, printing money.

What goes up can't come down

According to conventional wisdom, heart scan scores cannot be reduced.

In other words, say you begin with a heart scan score of 300. Conventional wisdom says you should take aspirin and a statin drug, eat a low-fat "heart healthy" diet, and take high blood pressure medications, if necessary.

If your heart scan score goes up in a year or two, especially at an annual rate of 20% or more, then you are at very high risk for heart attack. If the heart scan score stays the same, then your risk is much reduced. These observations are well-established.

But more than 99% of physicians will tell you that reducing your heart scan score is impossible. Don't even try: Heart scan scores can go up, but they can't go down.

Baloney. Heart scan scores can indeed go down. And they can go down dramatically.

It is true that, following conventional advice like taking a statin drug, following a low-fat diet, and taking aspirin will fail to reduce your heart scan score. A more rational approach that 1) identifies all causes of coronary plaque, 2) corrects all causes while including crucial strategies like omega-3 fatty acid supplementation, vitamin D supplementation, and thyroid function normalization, is far more likely to yield a halt or reduction in score.

While not everybody who undertakes the Track Your Plaque program will succeed in reducing their heart scan score, a growing number are enjoying success.

A small portion of our experience was documented this past summer. (I collected and analyzed the data with the help of Rush University nutrition scientist, Dr. Susie Rockway, and statistician, Dr. Mary Kwasny.)


Effect of a combined therapeutic approach of intensive lipid management, omega-3 fatty acid supplementation, and increased serum 25 (OH) vitamin D on coronary calcium scores in asymptomatic adults.

Davis W, Rockway S, Kwasny M.

The impact of intensive lipid management, omega-3 fatty acid, and vitamin D3 supplementation on atherosclerotic plaque was assessed through serial computed tomography coronary calcium scoring (CCS). Low-density lipoprotein cholesterol reduction with statin therapy has not been shown to reduce or slow progression of serial CCS in several recent studies, casting doubt on the usefulness of this approach for tracking atherosclerotic progression. In an open-label study, 45 male and female subjects with CCS of > or = 50 without symptoms of heart disease were treated with statin therapy, niacin, and omega-3 fatty acid supplementation to achieve low-density lipoprotein cholesterol and triglycerides < or = 60 mg/dL; high-density lipoprotein > or = 60 mg/dL; and vitamin D3 supplementation to achieve serum levels of > or = 50 ng/mL 25(OH) vitamin D, in addition to diet advice. Lipid profiles of subjects were significantly changed as follows: total cholesterol -24%, low-density lipoprotein -41%; triglycerides -42%, high-density lipoprotein +19%, and mean serum 25(OH) vitamin D levels +83%. After a mean of 18 months, 20 subjects experienced decrease in CCS with mean change of -14.5% (range 0% to -64%); 22 subjects experienced no change or slow annual rate of CCS increase of +12% (range 1%-29%). Only 3 subjects experienced annual CCS progression exceeding 29% (44%-71%). Despite wide variation in response, substantial reduction of CCS was achieved in 44% of subjects and slowed plaque growth in 49% of the subjects applying a broad treatment program.

Gretchen's postprandial diet experiment

Gretchen sent me the results of a little experiment she ran on herself. She measured blood glucose and triglycerides after 1) a low-fat diet and 2) a low-carb diet.









Gretchen describes her experience:

Several years ago I received a windfall of triglyceride strips that would expire in a week or so. I hated to waste them, so I decided to use them to test my triglyceride and BG responses to two different diets: low carb and low fat.

The first day I followed a low-fat diet. For breakfast I ate a lot of carbohydrate, including 1 oz of spaghetti cooked al dente and ¾ cup of white rice. For the rest of the day I ate less carbohydrate but continued to eat low fat.

The second day I followed a low-carb diet. For breakfast I ate a lot of fat, including a sausage, mushrooms fried in butter, 2 slices of bacon, and ¼ cup of the creamy topping of whole-milk yogurt. For the rest of the day I ate less fat, especially less saturated fat, but continued to eat low carb.

Both days I measured both BG and triglyceride levels every hour until I went to bed. On the low-carb day I had 3 meals. On the low-fat day, I was constantly hungry, had 4 meals, and kept snacking.

You can see the results in Figure 1. On the low-fat diet, after a “healthy” low-fat breakfast of low-glycemic pasta with low-fat sauce, my BG levels shot up to over 200 mg/dL and took more than 6 hours to come down. My triglycerides, however, remained low, and at first I thought perhaps the low-fat diet might be better overall. However, after about 6 hours, the triglyceride levels started to increase steadily, and by the next morning, they were higher than they had been the day before.
On the low-carb diet, my BG levels stayed low all day. However, after meals, the triglyceride levels skyrocketed. After meals they came down, and by the next morning they were lower than they had been the day before.

As I interpret these results, the high triglyceride levels after eating the high-fat meals represent chylomicrons, the lipoproteins that transport fat from your meals to the cells of your body. The high triglyceride levels the morning after eating the low-fat meals represent very low density lipoprotein, which takes the cholesterol your liver synthesizes when your intake of dietary cholesterol is low and distributes it to cells that need it, or again, to the fat for storage.

There are several interesting factors to consider here. First, when you have a lipid test done at the lab, it’s usually done fasting, which means first thing in the morning after not eating for 8 to 12 hours. It tells you nothing about what your triglyceride levels were all day.

Second, the low-carb diet resulted in lower fasting triglyceride levels, but much higher postprandial triglyceride levels. Which are more dangerous? I’m afraid I don’t know. You should also note that the high-fat, low-carb breakfast was extremely high in fat, including saturated fat. I don’t normally eat that much fat but wanted to test extremes.

Third, although the low-fat diet didn’t produce the very high postprandial triglyceride levels that the high-fat diet did, it produced extremely high BG levels that persisted for 6 hours. Some people think that it’s oxidized and glycated lipids that are the dangerous ones, so high BG levels and normal triglyceride levels might be more dangerous than very high triglyceride levels and normal BG levels. Note that high BG levels also contribute to oxidation rates.

Fourth, this shows the results of an experiment with a sample size of one. My physiology might not be typical. If you want to know how your own body’s lipids respond to different types of diets, you should get a lipid meter and test yourself. Unfortunately, your insurance is unlikely to want to pay for this, so it will be an expensive experiment.

The main point of this is that the results of different diets are complex. We have to eat. And what we eat can affect many different systems in our bodies. Finding the ideal diet that matches our own physiology and results in the best lipid levels as well as BG levels is a real challenge.



This was a lot of effort for one person. Thanks to Gretchen for sharing her interesting experience.

Gretchen makes a crucial point: Some of the effects of diet changes evolve over time, much as triglyceride levels changed substantially for her on the day following her experiment. Wouldn't it be interesting to see how postprandial patterns develop over time if levels were observed sequentially, day after day?

The stark contrast in blood sugars is impressive--Low-carb clearly has the advantage here. Are there manipulations in diet composition in low-carb meals that we can make to blunt the early (3-6 hour) postprandial lipoprotein (triglyceride) peak? That's a topic we will consider in future.

More of Gretchen's thoughts can be found at:

http://wildlyfluctuating.blogspot.com
http://www.healthcentral.com/diabetes/c/5068

After-eating effects: Carbohydrates vs. fats

In the ongoing debate over whether it's fat or carbohydrate restriction that leads to weight loss and health, here's another study from the Oxford group examining the postprandial (after-eating) effects of a low-fat vs. low-carbohydrate diet. (Roberts R et al, 2008; full-text here.)

High-carbohydrate was defined as 15% protein; 10% fat; 75% carbohydrate (by calories), with starch:sugar 70:30.

High-fat was defined as 15% protein; 40% fat; 45% carbohydrate, with starch:sugar 70:30. (Yes, I know. By our standards, the "high-fat" diet was moderate-fat, moderate-carbohydrate--too high in carbohydrates.)

Blood was drawn over 6 hours following the test meal.




Roberts R et al. Am J Clin Nutr 2008

The upper left graph is the one of interest. Note that, after the high-carbohydrate diet (solid circles), triglyceride levels are twice that occurring after the high-fat diet (open circles). Triglycerides are a surrogate for chylomicron and VLDL postprandial lipoproteins; thus, after the high-carbohydrate diet, postprandial particles are present at much higher levels than after the high-fat diet. (It would have been interesting to have seen a true low-carbohydrate diet for comparison.) Also note that, not only are triglyceride levels higher after high-carbohydrate intake, but they remain sustained at the 6-hour mark, unlike the sharper decline after high-fat.

It's counterintuitive: Postprandial lipoproteins, you'd think, would be plentiful after ingesting a large quantity of fat, since fat must be absorbed via chylomicrons into the bloodstream. But it's carbohydrates (and obesity, a huge effect; more on that in future) that figure most prominently in determining the pattern and magnitude of postprandial triglycerides and lipoproteins. Much of this effect develops by way of de novo lipogenesis, the generation of new lipoproteins like VLDL after carbohydrate ingestion.

We also see this in our Track Your Plaque experience. Rather than formal postprandial meal-testing, we use intermediate-density lipoprotein (IDL) as our surrogate for postprandial measures. A low-carbohydrate diet reduces IDL dramatically, as do omega-3 fatty acids from fish oil.
Look like Jimmy Stewart

Look like Jimmy Stewart


"This diet works great," Don declared. "But I think I've lost too much weight."

At 67 years old and 5 ft. 11 inches, Don began the program weighing 228 lbs (BMI 31.9). Because of high triglycerides, high blood sugar, high c-reactive protein, and excessive small LDL, I instructed Don to eliminate all wheat products from his diet, along with cornstarch and sweets. His intake of lean meats, eggs, vegetables, oils, raw nuts, etc. was unlimited.

Don now weighed 194 lbs, down 34 lbs over 6 months (BMI 27.1). Triglycerides, blood sugar, blood pressure, and well-being had improved dramatically; small LDL, however, had dropped only 30%--still room for improvement.

"My friends say I'm too skinny. They ask if I have cancer!"

I've heard this many times: Someone loses weight in a relatively short period of time and friends and family tell you you're too skinny. "It must be cancer. Nobody loses weight like that."

Unfortunately, many Americans have forgotten what normal looks like. Normal is certainly not a 190-lb, 5 ft 4 in woman, nor is it a 228 lb, 5 ft 11 inch man. But Americans have put on so much weight that the prevailing view of what constitutes "normal" weight has been revised upward. Normal is closer to what we see in old movies from the 1940s and '50s with people like Jimmy Stewart and Donna Reed. That's what we are supposed to look like.

So Don actually remains mildly overweight but is judged as "too skinny," or even cancer-ridden, by friends and family.

Ignore such comments. As you lose pounds and approach a truly desirable weight, realize that you are returning to the normal state, not the vision of "normal" now held by most Americans.

Comments (23) -

  • AllenS

    1/15/2010 8:40:24 PM |

    This is funny because as a 5'11" male I'm 165lbs and considered by some to be "emaciated" even though I have 10% body fat and quite a bit of muscle. I remember 45 years ago as a kid when my 6' tall father weighed 170lbs. Nobody ever called him skinny because he pretty much looked like all of his friends. He was considered normal at that time. I remember his weight at that time because he often boasted about it seeing as how he only weighed 125 lbs when he was drafted into the Navy.

    We have indeed forgotten what normal looks like.

  • Sarah

    1/15/2010 9:07:45 PM |

    I think you're onto something with this 'standards' business. I'm down to 171 pounds (nearly 70 pounds!) since going on my diet. It hasn't been a FAST loss, but people who haven't seen me in a while are surprised and remark that I look like a 'stick'.

    Since when did 171 fall into the 'stick' range for a 5'4" woman? Maybe >30 BMI is thin for Kentucky.

    Note: I love Jimmy Stewart!

  • Jeanie Campbell

    1/15/2010 10:32:44 PM |

    Excellent post!  My question, then, is, where do we find a reliable place to find out what our desirable weight IS?  I'm not sure I trust the ones I have found on-line.  Can you recommend one?  Especially for folks over 50.  Thanks!

  • whatsonthemenu

    1/15/2010 11:44:56 PM |

    "Unfortunately, many Americans have forgotten what normal looks like. Normal is certainly not a 190-lb, 5 ft 4 in woman, nor is it a 228 lb, 5 ft 11 inch man."

    So true.  Walking through the airport terminal on a visit from Asia immediately oriented me back to the US with the long chain of fast food franchises and big, waddling passengers.  A trip to Walmart to see morbidly obese people in motorized carts is a tourist attraction for Asians.  They can't believe it until they see it.

  • jnkdaniel@hotmail.com

    1/16/2010 1:16:58 AM |

    Yes, this blog is definitely detrimental to my fat.

    For five months, I've swam, taken fish oil, cut out juice and bread from my fridge.  As a result I've lost 16 pounds, 12 beats per minute, and 3 off my blood pressure.

    I'm currently 29m 6'2 and at 184 lb, 48 bpm resting, and at 125 for blood pressure.

    It is truly scary to see how easy it is to lose weight once you know how bad certain foods are.  It is borderline addicting!

    I'm curious to see if I will hit an equilibrium or I will have to do something to stop the weight loss once I reach 175-180.

  • Anonymous

    1/16/2010 2:01:02 AM |

    This is so true, many of my friends think I'm extremely skinny, yet I'm at my optimal weight. My mom refuses to lose more weight,she says "people will say that I look old and sick"

  • Anonymous

    1/16/2010 6:26:42 AM |

    You hit the nail on the head. I too, as a 50-something year old male, was about 220 at 5'10" last year this time, and as I approached 185 mid-year, several folks asked, "Are you all right?" and "Did you intend to lose the weight?" Yet I still am not at an ideal weight for my height, and although I look slim in comparison, I still have abdominal fat that needs to go. I've also had people tell me, "You look too thin," and "Don't lose any more weight." We must recapture a sense of normal. However, during a recent visit to a Glen Ivy Spa in So. Cal. my wife and I marveled at how many grossly obese people there were sauntering around in swimsuits. We've definitely got a problem here. For me, I'd rather look like Jimmie Stewart or Jack Lalane or Art Devany, and I don't care what anyone else thinks about it!

  • pmpctek

    1/16/2010 7:20:44 AM |

    I had a friend say to me once, "you lost a lot of weight, are you sick 'r something?"

    I'm a 5' 9" 49 y.o. North American male and went from 192 lbs. to 168 lbs. in nine months.  This was a couple years ago. I lost most of it off my mid-section and face.  I have the incredible shrinking waist (now 30 inches.)

    I did this by simply eliminating grains, starches, and sugars.  I actually had to slightly increase my daily calorie intake (than when I weighed 192) because I too was concerned I might have been losing too much weight.

    When I share with family and friends why I look so lean, that it's from permanent grain, starch and sugar abstention, they always respond with "oh no, I can't do that"  or "how can you do that?"

  • Kurt

    1/16/2010 1:36:58 PM |

    This is reassuring. I've been worrying because, since I started a heart healthy diet, I've gone from 183 lbs to 167 lbs, which is less than I weighed when I was 18 years old (170).

  • Dr. William Davis

    1/16/2010 3:00:21 PM |

    Hi, Jeannie--

    There are a number of ways to determine ideal weight. BMI, though an imperfect concept, is a good starting place. Here's a BMI calculator: http://www.nhlbisupport.com/bmi/

    This gives me an idea for a future post: "What is ideal weight?"

  • Aaron Blaisdell

    1/16/2010 4:20:20 PM |

    I won't even tell you what my Chinese in-laws think. Two English words my Chinese-speaking mother-in-law knows are "eat more." I always fend her of with with the retort "Che bao la."

  • Eclecbit

    1/16/2010 6:07:27 PM |

    There's also the problem of finding clothes that fit. I'm a 5'11" male and weigh 152lbs. I've got a 32" waist, but when I try on 32" waist pants they fall off of me because they're really 34" (I believe this is called vanity sizing), so I look for the 30" waist pants and guess what? There are none!

    Maybe it's because I live in the South, but 30" waist pants are pretty much non-existent, and the ones that I do find are always too short.

    My wife used to think I was too skinny, but then she remembered all her Oriental friends back in California who are as skinny as I am. For them it's considered normal.

  • Steve L.

    1/16/2010 6:26:22 PM |

    I say revel in it!  I knew from past temporary weight loss that people would start to notice after I lost 30 pounds or so.  Since I needed to lose 70 to get to ideal weight, I also knew that those comments were nothing but signs of sucess.  We truly have adapted to a new normal in our perceptions.  The shock value does diminish over time though.  Now three years out from adopting a healthy diet (currently 6'3", 190 lbs.), I got all the comments along the way, but now people have adjusted to my new look (as have I).  Once in a while though, I see someone, usually business-related, that I haven't seen for a few years, and they're shocked.  I just enjoy it, and try to recruit them over from the dark side.

    The thing that I find interesting now is that, while I was losing people were interesting in why I was losing, and several adopted the low-carb/paleo approach with great success.  But now that I have reached an ideal weight, the memory of the previous me fades, and few see me as a potential source of healthy diet information.  I think some actually think I must be a bit of a freak for having done so well, and so there's nothing useful to be learned from me by non-freaks.

  • Anne

    1/16/2010 9:36:40 PM |

    I am another who lost weight when I dropped all grains and sugars and greatly limited high carb veges and fruits. The weight just melted away. I did not need to lose much and when I hit 20 lbs, the weight loss stopped. I have been at 120-125 for many months now. I am 5' 4". I never feel hungry eating the higher fat diet. Honestly, sometimes I do miss the junky food but not enough to eat it and jeopardize my health.

  • Nick

    1/17/2010 3:38:15 AM |

    I wonder if anyone has information on cornstarch and why it places right next to wheat as a 'food' to avoid?  I have seen a great deal of convincing argument with regard to wheat, but almost none with regard to cornstarch (other than for those who may need to closely watch blood glucose levels).  

    If anyone can lead me to more information on how it affects our organism, I would great appreciate it.

  • steve

    1/17/2010 4:21:28 PM |

    Dr. Davis.  If you do a post on ideal weight, it might be helpful to include a discussion of muscle mass.  Many athletes are heavier than those of comparable ages in the general population, but have body fat levels that are extremely low.  There is a trade-off with weight loss and muscle loss, and I suppose a happy equilibrium at some leve.  Perhaps body fat level is a better gauge than absolute weight level, but hard to accuratley measure.  Thanks,

  • Claire

    1/18/2010 6:40:22 AM |

    I read an newspaper article about how parents in the UK didn't realise their children were obese. Yes, that's obese - not just overweight.

    We have lost sight of what it is to be of normal weight. I catch myself looking at people's sizes in old movies to remind myself of what "normal" should be.

  • AllenS

    1/18/2010 5:43:56 PM |

    I really don't like the BMI indicator. First, there is no differentiation between males and females or body type. Fit males who have any kind of muscle tone or who may be big-boned will invariably have a BMI greater than 25. I'm very close even though I'm only 10% body fat.

    Instead, I think that a better measure is to ignore weight altogether and get your % body fat computed. Ideally it should be 14-17% for males and 21-24% for females.

    I too have difficulty finding pants that fit. I wear a 30" waist. Its tough to find anything smaller than a 38"-40" waist which is pretty sad.

  • Anonymous

    1/18/2010 11:57:30 PM |

    Based on the posts here on HeartScan and my brothers insistence Atkin's was his preferred effective weight control solution, I started eating meat again after 10 years of being a pescatarian. I put on 12 lbs in 3 months.  OK, I am not too keen on eating slabs of meat and may have gone overboard with sausage meat / chicken wings but I hope my next blood test will show an increase in HDL as a result of the added fat and lower wheat/grains

    BTW. I stopped my 20mgs crestor and got a base line several months back (too scary !). I have taken 20mgs and 40mgs crestor with the latter leading to some muscle pain but perfect LDL (60). HDL only went up with Niacin (31 to 45 )

    What I want is no more than 20mgs crestor (which gives me LDL circa 75 and I can tolerate well) and to elevate my HDL to 60 without having to eat raw cow.

    This site is a great resource. I would like to see Dr D square off against the celeb TV Dr Oz who pushes high grain diets and low saturated fat.
    Trev

  • Apolloswabbie

    1/30/2010 10:03:43 PM |

    I think some of the response to too skinny is because folks are faced with how 'not skinny' they are looking at those who are not.

  • Anonymous

    2/8/2010 10:14:07 PM |

    I'm a caucasian male, 6'2" and I've been healthily below 160.  I have a thin body.  I don't know if it's because my bones are smaller, or what, but this is normal for me.

    And, I feel for the thin folks in the south.  When we lived in TN for a few years, I had a heck of a time finding 32" waist pants.  Now that I'm back in CA, it's much easier.

  • lockeender

    5/6/2010 4:09:25 AM |

    Jimmy Stewart was thought too skinny by Hollywood and the Army at the time.  When he was first signed to MGM they recognized that Stewart had an uncanny screen charisma and great star potential, but they considered him just so goofy looking that they didn't buy him having any male star sex appeal.  MGM wanted someone to compete with Tyrone Power, Clark Gable, Spencer Tracy and up and comers like Cary Grant (Grant would be a better example for you than Stewart).  Before MGM ever put Stewart in a movie they put him with one of the studio weight trainers, hoping to add some muscle to his physique.  The trainer had Stewart lifting weights and drinking a gallon of milk everyday.  After a month of this regimen Stewart had gained about three pounds, mostly of bloat.  MGM put him in a variety of bit parts but they figured he was basically useless to them so they loaned him out to Columbia for a pair of pictures, You Can't Take it with You and Mr. Smith Goes to Washington.  Stewart's star was made and he returned to MGM to make a slew of great films, Destry rides again, Philadelphia Story, & The shop Around the corner.  Stewart came from a very patriotic, midwestern family.  in 1940 Stewart basically quit his studio contract (after filming A Mortal Storm) and recognizing the world situation, he went to enlist in the Army with the idea of entering the Air Corps to train as a pilot.  He was rejected flat out because he did not weigh enough for the minimum standard to enlist.  And Stewart was 6' 3&3/4" he weighed next to nothing!  Since he was only a few lbs under, Stewart went back the next week, this time after waterloading himself.  he barely made it through the physical before bursting, but he was able to eek over that minimum weight standard by a single pound.  By the time Pearl Harbor hit, Stewart was a certified pilot and he spent most of the war continually flying bombing missions over Europe.

    Cary Grant on the other hand, would be a superb example. Grant began life as a circus tumbler, and he maintained his athleticism throughout his life.  His remarkable lack of aging until his final decade was due to his  eschewing alcohol and smoking in his private life, which was both very rare at the time and ironic considering the suave characters he played always drank and smoked.  He may also have been one of the oddball anti-sugar hollywood types (Gloria Swanson was one) that refused to eat anything with sugar in it.  But I'm not certain on that.

  • buy jeans

    11/3/2010 3:43:15 PM |

    Ignore such comments. As you lose pounds and approach a truly desirable weight, realize that you are returning to the normal state, not the vision of "normal" now held by most Americans.

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