Rerun: To let low-carb right, you must check POSTPRANDIAL blood sugars

2. April 2010 William Davis (12)
Checking postprandial (after-eating) blood sugars yields extraordinary advantage in creating better diets for many people.

This idea has proven so powerful that I am running a previous Heart Scan Blog post on this practice to bring any newcomers up-to-date on this powerful way to improve diet, lose weight, reduce small LDL, reduce triglycerides, and reduce blood pressure.



To get low-carb right, you need to check blood sugars

Reducing your carbohydrate exposure, particularly to wheat, cornstarch, and sucrose (table sugar), helps with weight loss; reduction of triglycerides, small LDL, and c-reactive protein; increases HDL; reduces blood pressure. There should be no remaining doubt on these effects.

However, I am going to propose that you cannot truly get your low-carb diet right without checking blood sugars. Let me explain.

Carbohydrates are the dominant driver of blood sugar (glucose) after eating. But it's clear that we also obtain some wonderfully healthy nutrients from carbohydrate sources: Think anthocyanins from blueberries and pomegranates, vitamin C from citrus, and soluble fiber from beans. There are many good things in carbohydrate foods.

How do we weigh the need to reduce carbohydrates with their benefits?

Blood sugar after eating ("postprandial") is the best index of carbohydrate metabolism we have (not fasting blood sugar). It also provides an indirect gauge of small LDL. Checking your blood sugar (glucose) has become an easy and relatively inexpensive tool that just about anybody can incorporate into health habits. More often than not, it can also provide you with some unexpected insights about your response to diet.

If you’re not a diabetic, why bother checking blood sugar? New studies have documented the increased likelihood of cardiovascular events with increased postprandial blood sugars well below the ranges regarded as diabetic. A blood sugar level of 140 mg/dl after a meal carries 30-60% increased (relative) risk for heart attack and other events. The increase in risk begins at even lower levels, perhaps 110 mg/dl or lower after-eating.

We use a one-hour after eating blood sugar to gauge the effects of a meal. If, for instance, your dinner of baked chicken, asparagus brushed with olive oil, sauteed mushrooms, mashed potatoes, and a piece of Italian bread yields a one-hour blood sugar of 155 mg/dl, you know that something is wrong. (This is far more common than most people think.)

Doing this myself, I have been shocked at the times I've had an unexpectedly high blood sugar from seemingly "safe' foods, or when a store- or restaurant-bought meal had some concealed source of sugar or carbohydrate. (I recently had a restaurant meal of a turkey burger with cheese, mixed salad with balsamic vinegar dressing, along with a few bites of my wife's veggie omelet. Blood sugar one hour later: 127 mg/dl. I believe sugar added to the salad dressing was the culprit.)

You can now purchase your own blood glucose monitor at stores like Walmart and Walgreens for $10-20. You will also need to purchase the fingerstick lancets and test strips; the test strips are the most costly part of the picture, usually running $0.50 to $1.00 per test strip. But since people without diabetes check their blood sugar only occasionally, the cost of the test strips is, over time, modest. I've had several devices over the years, but my current favorite for ease-of-use is the LifeScan OneTouch UltraMini that cost me $18.99 at Walgreens.

Checking after-meal blood sugars is, in my view, a powerful means of managing diet when reducing carbohydrate exposure is your goal. It provides immediate feedback on the carbohydrate aspect of your diet, allowing you to adjust and tweak carbohydrate intake to your individual metabolism.

LDL glycation

1. April 2010 William Davis (11)
The proteins of the body are subject to the process of glycation, modification of protein structures by glucose (blood sugar). In the last Heart Scan Blog post, I discussed how glycated hemoglobin, available as a common test called HbA1c, can serve as a reflection of protein glycation (though it does not indicate actual Advanced Glycation End-products, or AGEs, just a surrogate indicator).

There is one very important protein that is subject to glycation: Apoprotein B.

Apoprotein B, or Apo B, is the principal protein of VLDL and LDL particles. Because there is one Apo B molecule per VLDL or LDL particle, Apo B can serve as a virtual VLDL/LDL particle count. The higher the Apo B, the greater the number of VLDL and LDL particles.

Because Apo B is a protein, it too is subject to the process of glycation. The interesting thing about the glycation of Apo B is that its "glycatability" depends on LDL particle size: The smaller the LDL particle, the more glycation-prone the Apo B contained within.

Younis et al have documented an extraordinary variation in glycatability between large and small LDL, with small LDL showing an 8-fold increased potential.

Think about it: Carbohydrates in the diet, such as wheat products and sugars, trigger formation of small LDL particles. Small LDL particles are then more glycation-prone by up to a factor of 8. Interestingly, HbA1c is tightly correlated with glycation of Apo B. Diabetics with high HbA1c, in particular, have the greatest quantity of glycated Apo B. They are also the group most likely to develop coronary atherosclerosis, as well as other consequences of excessive AGEs.

No matter how you spin it, the story of carbohydrates is getting uglier and uglier. Carbohydrates, such as those in your whole grain bagel, drive small LDL up, while making them prone to a glycating process that makes them more likely to contribute to formation of coronary atherosclerotic plaque.

High HbA1c: You're getting older . . . faster

28. March 2010 William Davis (16)
Over the years, we all accumulate Advanced Glycation End-products, or AGEs.

AGEs are part of aging; they are part of human disease. AGEs are the result of modification of proteins by glucose. AGEs form the basis for many disease conditions.

Accumulated AGEs have been associated with aging, dementia, cataracts, osteoporosis, deafness, cancer, and atherosclerosis. Most of the complications of diabetes have been attributable to AGEs.

There's one readily available method to assess your recent AGE status: HbA1c.

Hemoglobin is the oxygen-carrying protein of red blood cells. Like other proteins, hemoglobin becomes glycated in the presence of glucose. Hemoglobin glycation increases linearly with glucose: The higher the serum or tissue glucose level, the more glycation of hemoglobin develops. Glycated hemoglobin is available as the common test, HbA1c.

Ideal HbA1c is 4.5% or less, i.e., 4.5% of hemoglobin molecules are glycated. Diabetics typically have HbA1c 7.0% or greater, not uncommonly greater than 10%.

In other words, repetitive and sustained high blood glucose leads to greater hemoglobin glycation, higher HbA1c, and indicates greater glycation of proteins in nerve cells, the lens of your eye, proteins lining arteries, and apoprotein B in LDL cholesterol particles.

If AGEs accumulate as a sign of aging, and high blood sugars lead to greater degrees of glycation, it only follows that higher HbA1c marks a tendency for accelerated aging and disease.

Indeed, that is what plays out in real life. People with diabetes, for instance, have kidney failure, heart disease, stroke, cataracts, etc. at a much higher rate than people without diabetes. People with pre-diabetes likewise.

The higher your HbA1c, the greater the degree of glycation of other proteins beyond hemoglobin, the faster you are aging and subject to all the phenomena that accompany aging. So that blood glucose of 175 mg/dl you experience after oatmeal is not a good idea. 

The lesson: Keep HbA1c really low. First, slash carbohydrates, the only foods that substantially increase blood glucose. Second, maintain ideal weight, since normal insulin responsiveness requires normal body weight. Third, stay physically active, since exercise and physical activity exerts a powerful glucose-reducing effect. Fourth, consider use of glucose-reducing supplements, an issue for another day.

While HbA1c cannot indicate cumulative AGE status, it can reflect your recent (preceding 60 to 90 days) exposure to this age-accelerating thing called glucose.

If your doctor refuses to accommodate your request for a HbA1c test, you can perform your own fingerstick test.

Slash carbs . . . What happens?

26. March 2010 William Davis (20)
Cut the carbohydrates in your diet and what sorts of results can you expect?

Carbohydrate reduction results in:

Reduced small LDL--This effect is profound. Carbohydrates increase small LDL; reduction of carbohydrates reduce small LDL. People are often confused by this because the effect will not be evident in the crude, calculated (Friedewald) LDL that your doctor provides.

Increased HDL--The HDL-increasing effect of carbohydrate reduction may require 1-2 years. In fact, in the first 2 months, HDL will drop, only to be followed by a slow, gradual increase. This is the reason why, in a number of low-carb diet studies, HDL was shown to be reduced.--Had the timeline been longer, HDL would show a significant increase.

Decreased triglycerides--Like reduction of small LDL, the effect is substantial. Triglyceride reductions of several hundred milligrams are not at all uncommon. In people with familial hypertriglyceridemia with triglyceride levels in the thousands of milligrams per deciliter, triglyceride levels will plummet with carbohydrate restriction. (Ironically, conventional treatment for familial hypertriglyceridemia is fat restriction, a practice that can reduce triglycerides modestly in these people, but not anywhere near as effectively as carbohydrate restriction.) Triglyceride reduction is crucial, because triglycerides are required by the process to make small LDL--less triglycerides, less small LDL.

Decreased inflammation--This will be reflected in the crude surface marker, c-reactive protein--Yes, the test that the drug industry has tried to convince you to take statins drugs to reduce. In my view, it is an absurd notion that you need to take a drug like Crestor to reduce risk associated with increased CRP. If you want to reduce CRP to the floor, eliminate wheat and other junk carbohydrates. (You should also add vitamin D, another potent CRP-reducing strategy.)

Reduced blood pressure--Like HDL, blood pressure will respond over an extended period of months to years, not days or weeks. The blood pressure reduction will be proportion to the amount of reduction in your "wheat belly."

Reduced blood sugar--Whether you watch fasting blood sugar, postprandial (after-meal) blood sugars, or HbA1c, you will witness dramatic reductions by eliminating or reducing the foods that generate the high blood sugar responses in the first place. Diabetics, in particular, will see the biggest reductions, despite the fact that the American Diabetes Association persists in advising diabetics to eat all the carbohydrates they want. Reductions in postprandial (after-eating) blood sugars, in particular, will reduce the process of LDL glycation, the modification of LDL particles by glucose that makes them more plaque-causing.


You may notice that the above list corresponds to the list of common plagues targeted by the pharmaceutical industry: blood pressure, diabetes (diabetes being the growth industry of the 21st century), high cholesterol. In other words, high-carbohydrate, low-fat foods from the food industry create the list of problems; the pharmaceutical industry steps in to treat the consequences.

In the Track Your Plaque approach, we focus specifically on elimination of wheat, cornstarch, and sugars, the most offensive among the carbohydrates. The need to avoid other carbohydrates, e.g., barley, oats, quinoa, spelt, etc., depends on individual carbohydrate sensitivty, though I tend to suggest minimal exposure.

Normal fasting glucose with high HbA1c

23. March 2010 William Davis (24)
Jonathan's fasting glucose: 85 mg/dl
His HbA1c: 6.7%

Jonathan's high HbA1c reflects blood glucose fluctuations over the preceding 60-90 days and can be used to calculate an estimated average glucose (eAG) with the following equation:

eAG = 28.7 X A1c – 46.7

(For glucose in mmol/L, the equation is eAG = 1.59 × A1C - 2.59)

Jonathan's HbA1c therefore equates to an eAG of 145.59 mg/dl--yet his fasting glucose value is 85 mg/dl. 

This is a common situation: Normal fasting glucose, high HbA1c. It comes from high postprandial glucose values, high values after meals. 

It suggests that, despite having normal glucose while fasting, Jonathan experiences high postprandial glucose values after many or most of his meals. After a breakfast of oatmeal, for instance, he likely has a blood glucose of 150 mg/dl or greater. After breakfast cereal, blood glucose likely exceeds 180 mg/dl. With two slices of whole wheat bread, glucose likewise likely runs 150-180 mg/dl. 

The best measure of all is a postprandial glucose one hour after the completion of a meal, a measure you can easily obtain yourself with a home glucose meter. Second best: fasting glucose with HbA1c.

Gain control over this phenomenon and you 1) reduce fasting blood sugar, 2) reduce expression of small LDL particles, and 3) lose weight.  

Can you handle fat?

21. March 2010 William Davis (19)
No question: Low-carbohydrate diets generate improved postprandial lipoprotein responses.

Here's a graph from one of Jeff Volek's great studies:



Participants followed a low-carb diet of less than 50 g per day carbohydrate ("ketogenic") with 61% fat.   The curves were generated by administering a 123 g fat challenge with triglyceride levels assessed postprandially. The solid line represents the postprandial response at the start; dotted line after the 6-week low-carb effort.

Note that:

1) The postprandial triglyceride (area-under-the-curve) response was reduced by 29% in the low-carb diet.  That's a good thing.

2) The large fat challenge generated high triglycerides of greater than 160 mg/dl even in the low-carb group. That's a bad thing. 

In other words, low-carb improves postprandial responses substantially--but postprandial phenomena still occur. Postprandial triglycerides of 88 mg/dl or greater are associated with greater heart attack risk because they signify the presence of greater quantities of atherogenic (plaque-causing) postprandial lipoproteins.

A full discussion of these phenomena can be found in the Track Your Plaque Special Report, Postprandial Responses: The Storm After the Quiet!, part of a 3-part series on postprandial phenomena.

Statin stupid

19. March 2010 William Davis (0)
If we followed the lead of the pharmaceutical industry and my cardiology colleagues, we would all subscribe to the "statins for all" philosophy. There is now $2 billion of clinical "research" to back up this "evidence-based" practice.

I do not endorse this "statins for all" philosophy. I believe it is a product of the raw profiteering of the pharmaceutical industry, who are adept at recruiting physicians to their cause.

But lost in the confusion of tainted studies and over-the-top media saturation is the fact that there are small groups of people who likely do obtain benefit from statin drugs. They would certainly benefit from better informed scrutiny of their lipoprotein and metabolic abnormalities. But treatment may involve statins.

This is entirely distinct from the "statins for all" argument, the simpleminded rule that primary care physicians and cardiologist are told to follow.

Groups who may indeed benefit from statin therapy include:

Homozygous or heterozygous familial hypercholesterolemia--Lacking a receptor for LDL particles, LDL piles up to very high levels in these people. LDLs of 300+ are common and lead to heart disease and stroke at relatively young ages.

Combined mixed hyperlipidemia--Among the one or more genetic defects underlying this condition involves excessive production of apoprotein B and VLDL particles. This leads to high risk for heart disease.

People unable to follow a diet to correct their lipid disorder--I have 80+-year old patients, for instance, who say, "I've eaten this way for 82 years. I'm not going to change now!" In the absence of diet and other efforts (e.g., omega-3 fatty acids from fish oil), drugs may be the answer.

In other words, of the $27 billion annual bill for statin drugs, perhaps a tiny fraction is truly necessary. The majority of people taking statin drugs would not really need them if they had the real answers. But don't let that confuse us: There are some people who do indeed benefit.

Butter and insulin

19. March 2010 William Davis (83)
In a previous post, Atkins Diet: Common Errors, I commented on butter's unusual ability to provoke insulin responses. I offer this as a possible reason why, after a period of effective weight loss on a low-carbohydrate program, inclusion of some foods, such as butter, will trigger weight gain or stall weight loss efforts.

This develops because of butter's insulin-triggering effect, doubling or tripling insulin responses (postprandial area-under-the-curve). If insulin is triggered, fat gain follows.

Here's one such study documenting this effect: Distinctive postprandial modulation of ß cell function and insulin sensitivity by dietary fats: monounsaturated compared with saturated fatty acids

López et al 2008


From Lopez et al 2008. Mean (± SD) plasma glucose, insulin, triglyceride, and free fatty acid (FFA) concentrations during glucose and triglyceride tolerance test meal (GTTTM) with no fat (control), enriched in monounsaturated fatty acids (MUFAs) from refined olive oil (ROO meal), with added butter, with a mixture of vegetable and fish oils (VEFO) or with high-palmitic sunflower oil (HPSO). N = 14.

The postprandial (after-eating) area-under-the-curve is substantially greater when butter is included in the mixed composition meal. This effect is not unique to butter, but is shared by most other dairy products.

Fat, in general, does not make you fat. But butter makes you fat.

Vitamin D as a cardiovascular risk factor gains ground

18. March 2010 William Davis (0)
If you were reading The Heart Scan Blog back in 2007, or read my Life Extension article on vitamin D deficiency as a cardiovascular risk factor, you already knew that vitamin D deficiency is rampant and adds to cardiovascular risk.

Results of a study from the Intermountain Medical Center Heart Institute in Utah bolster the concept that vitamin D deficiency is a cardiovascular risk factor, vitamin D normalization/supplementation reduces cardiovascular risk.

Science Daily reported:

For the first study, researchers followed two groups of patients for an average of one year each. In the first study group, over 9,400 patients, mostly female, reported low initial vitamin D levels, and had at least one follow up exam during that time period. Researchers found that 47 percent of the patients who increased their levels of vitamin D between the two visits showed a reduced risk for cardiovascular disease.


In the second study, researchers placed over 31,000 patients into three categories based on their levels of vitamin D. The patients in each category who increased their vitamin D levels to 43 nanograms per milliliter of blood or higher had lower rates of death, diabetes, cardiovascular disease, myocardial infarction, heart failure, high blood pressure, depression, and kidney failure. Currently, a level of 30 nanograms per milliliter is considered "normal."


Over the past 4 years, people in our program have been enjoying the extravagant benefits of vitamin D restoration. Cardiovascular benefits are becoming better documented and the bone health, cancer-preventing, insulin-normalizing, mood-adjusting, and anti-inflammatory effects likewise.

Atkins Diet: Common errors

18. March 2010 William Davis (74)
No doubt: The diet approach advocated by the late Dr. Robert Atkins was a heck of a lot closer to an ideal diet than the knuckleheaded advice emitting from the USDA, American Heart Association, American Diabetes Association, and the Surgeon General's office.

But having just spent a week with Atkins low-carbers, here are some common errors that I see many make, errors that I believe have long-term health consequences, including impairment of weight loss.

Excessive consumption of animal products--Non-restriction of fat often leads to over-reliance on animal products. Higher intakes of red meats (heme proteins?) have been strongly associated with increased risk for colon and other gastrointestinal tract cancers. It is not a fat issue; it is an animal product issue. We should consume less meat, more vegetables and other plant-sourced foods.

Consumption of cured meats--Cured, processed meats, such as sausage, hot dogs, salami, bologna, and bacon, have a color fixative called sodium nitrite, an additive that has been confidently linked to gastrointestinal cancers. Risk is likely dose-dependent: The more you ingest, the greater the long-term risk.

Overconsumption of dairy products--Dairy products, especially milk, yogurt, cottage cheese, and butter, are potent insulinotropic foods, i.e., foods that trigger insulin release. There can be up to a tripling of insulin (area-under-the-curve) levels. This is not good in a world populated with tired, overworked pancreases, exhausted from a lifetime of high-carbohydrate eating.

Too many calories--While I agree that "a calorie is a calorie" and "calories in, calories out" are faulty concepts, I have anecdotally observed that long-time low-carbers often trend towards unlimited consumption of food, a phenomenon that seems to result in weight gain, especially in the sedentary. I wonder if this is a reflection of the insulinotropic action of dairy products and other proteins, compounded by the poor insulin responsiveness that develops with lack of physical activity. Factor into this conversation that lower calorie intake extends life, probably substantially (Sirt-2 activation and related phenomena, a la resveratrol). If lower calorie intake extends life, unlimited calorie intake likely shortens life.

Please don't hear this as low-carb bashing--it is not. It is a call to improve diets and not stumble into common traps that can impair heart health, weight loss, and longevity.
Noodles without the headaches

Noodles without the headaches

14. October 2010 William Davis (26)
If you are looking for a wheat-free noodle or pasta, shirataki noodles are worth a try.

Shirataki noodles are low-carbohydrate (less than 3 g per 8 oz package) and, of course, do not trigger all the unhealthy effects of wheat--no blood sugar/insulin provocation, no addictive brain effects (exorphins), no gluten-mediated inflammatory effects.

(I advise avoiding gluten-free pasta alternatives made with rice flour and other common gluten alternatives, since they trigger blood sugar, small LDL, and growth of visceral fat just like wheat.)

I made a stir-fry using the shirataki-tofu noodles, shown below. (Tofu is added to make the noodles more noodly in consistency, as opposed to the chewier non-tofu variety.) The noodles were a lot like the ramen I used to eat as a kid. They were filling and tasted great in the sesame oil, soy sauce, tofu, and vegetables I used.


The noodles are easy to use. Just drain liquid out of package. (The noodles come in water.) Rinse in collander 30 seconds, then boil for 3 minutes. Add to your stir-fry or other dish. Some manufacturers, such as House Foods, also have angel hair and fettucine style noodles.
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Comments (26) -

  • Kathryn

    10/14/2010 3:05:32 PM |

    Well, since you touch on it, i'd be very interested in what you think about GF alternative flours.

    I know you have said in various places to avoid wheat & "corn starch."  Also indications that oats are not very good for us either.  But there are so many other grains.  Amaranth & quinoa are both supposed to be high in protein. Buckwheat?  Corn meals?  Millet?  I could go on & on.

    Are nut & coconut flours the only option to living low carb?  

    I try to keep your recommendations in mind, but frankly, i struggle with giving up all grains.

    Thanks.

  • Kent

    10/14/2010 3:13:18 PM |

    Dr Davis, I certainly appreciate you looking for alternatives to the destruction wheat poses, but seems like this product may have issues as well?

    Wouldn't the problems with soy greatly out weigh anything positive one could gain from using tofu?

    This was taken from an article on Dr. Mercola's site concerning tofu. http://articles.mercola.com/sites/articles/archive/2008/09/18/what-s-so-bad-about-tofu.aspx

    "Many health-conscious Americans, in an effort to improve their eating habits, have switched to eating tofu in place of meat or eggs. The soy industry would have you believe that this is a smart move for your heart health, but in reality processed soy, which includes tofu, is not a health food.

    You are much better off eating organic eggs, grass-fed meat and raw dairy products than you are eating processed soy.

    "Unlike in Asia where people eat small amounts of whole soybean products, western food processors separate the soybean into two golden commodities--protein and oil. There's nothing safe or natural about this,” Dr. Daniel says.

  • Nancy

    10/14/2010 3:18:11 PM |

    To Kent:  shirataki noodles are available WITHOUT soy, but you have to order those online, mostly the grocery store variety has soy.  The ones from MiracleNoodle.com are soy free.  I buy mine online at amazon.com, try this link: http://www.amazon.com/JFC-White-Shirataki-Noodles-16-0/dp/B002GDH5Y8/ref=sr_1_6?ie=UTF8&s=grocery&qid=1287069415&sr=1-6

  • Anna

    10/14/2010 3:31:32 PM |

    I don't miss noodles enough to bother with these, but I have tried them.  They work best with Asian-style noodle dishes better than Italian-style dishes, IMO.  I used the noodles made without tofu, however, as I make antibodies to soy, so I avoid it.

    One thing to note for those who try these noodles for the first time - when the package is first opened the aroma is slightly fishy.  They are NOT spoiled.  The smell will go away when they are drained and rinsed.  I tossed two packages before I discovered that was normal.  

    Dana Carpender, a popular low carb cookbook author and blogger, had a post up on her Hold The Toast blog not long ago about these noodles.

    Kathryn,

    Quinoa, and maybe amaranth (I haven't looked at the protein content of that one)  IS higher in protein (and the protein is more complete) than the common grains, but I wouldn't say it is HIGH in protein.  It still has a considerable amount of starch.  That's probably fine if for those who have a robust glucose tolerance.   But I need to limit starches, so I still limit these foods.  I might toss in a handful of quinoa to a pot of stew to thicken it up, but per serving, that's not much at all.  

    The longer I cook for my family with little or no without grains, the less I miss them.   I'd like my son to grow up without a huge craving for grain foods.

  • Marc

    10/14/2010 3:35:43 PM |

    Anna, you beat me to the punch.

    I call these noodles "FISH STRINGS"
    They smell bad Wink

    I guess it's better then pasta, but all in all, it's still a pretty processed product.

    Marc

  • Nancy

    10/14/2010 4:03:14 PM |

    Try Kelp Noodles sometime. I get them at Whole Foods, in the Deli case (refrigerated).  They're virtually tasteless and when cooked have a great noodle texture.

    I much prefer them to Shirtaki noodles.

  • Hans Keer

    10/14/2010 4:24:26 PM |

    Sorry doc, But now you avoid the gluten from grains and you introduce the lectins from the legume soy. This leads to a leaky gut and autoimmune diseases http://bit.ly/a9Gvjk

  • malpaz

    10/14/2010 5:28:28 PM |

    soy noodles??!?! tofu....for real?

  • Tommy

    10/14/2010 6:27:24 PM |

    "Unlike in Asia where people eat small amounts of whole soybean products, western food processors separate the soybean into two golden commodities--protein and oil. There's nothing safe or natural about this,” Dr. Daniel says."

    While I agree with the soy issue and stay away from processed soy (I do eat a little fermented soy tempeh) I am not sure about these reports I've read about the Japanese eating soy in small amounts. A Japanese friend of mine who recently came back to the U.S. after living in Japan for a few years says that there are actually Tofu stands on the street much like hot dog vendors in the U.S. He says they have tofu of all kinds (even flavored) and it is a popular snack. According to him Tofu is everywhere in Japan. He was puzzled when I mentioned the reports of low soy/tofu consumption in Asia.

  • Anonymous

    10/14/2010 7:03:38 PM |

    I tried shirataki noodles for the first time and loved them. I agree Asian dishes would be absolutely delightful with these noodles.

    @Kent. I have heard a fair amount about this Dr. Mercola you speak of. Sounds like he's not the most sciencific guy out there. Is he just out there trying to sell his products and really not caring about the science part of it?

    See below:
    http://www.sciencebasedmedicine.org/?p=2116

  • Nancy

    10/14/2010 7:44:14 PM |

    Dr Mercola is one of the only sane voices out there and he is right about soy.  And yes he IS science based, the difference is he tells the truth and doesn't hide the truth and just dole out pills like most doctors.

  • Tommy

    10/14/2010 8:30:57 PM |

    http://www.quackwatch.com/11Ind/mercola.html

  • Nancy

    10/14/2010 9:07:18 PM |

    of course the government tries to silence Dr Mercola, if they knew about the Heart Scan blog they'd try to silence its author as well since it doesnt recommend the food pyramid and tons of grains to support the US dept of Agriculture, LOL.  Its so obvious.

  • Kathryn

    10/14/2010 9:39:32 PM |

    Tommy, if you are interested in good, alternative medicine & natural ways of healing, Quackwatch is NOT the place to get your info.  He is paid much money to present his very biased (in favor of conventional medicine) articles.  

    If he doesn't yet have an article on what Dr. Davis does here, he probably soon will.

  • Dr. William Davis

    10/14/2010 10:03:30 PM |

    Let me add a qualifying comment.

    This, and perhaps some other ideas and suggestions in future, are simply meant to help people who seek replacements for familiar wheat-based foods.

    However, I believe that we should still focus primarily on real foods, not substitutes. Real eggs, real meats, real vegetables, real nuts, etc.

    Foods like shirataki noodles are meant to be occasional fun dishes.

    For the majority of people, I do not share Joe Mercola's fear of soy, provided you take an iodine source such as kelp tablets.

  • Dr. William Davis

    10/14/2010 10:04:43 PM |

    Hi, Kathryn--

    Gluten-free foods are candy, unfortunately.

    Here's my previous post on this issue: http://heartscanblog.blogspot.com/2010/07/what-increases-blood-sugar-more-than.html

  • Anonymous

    10/14/2010 10:05:48 PM |

    Quackwatch busted:  http://www.gaia-health.com/articles251/000277-quackbusters-are-busted.shtml

  • Tommy

    10/14/2010 10:21:28 PM |

    I have never read or followed Quackwatch and have no interest. While Googling Mercola that popped up. Ionly  posted it only as a statement that for every claim of life saving/altering advice one can point out there are just as many who disagree with it and show data to support their disagreements with all these doctors and gurus, diets etc.

    I continue to be amazed at the support "both" sides of all this diet stuff show; all with supporting data and studies.  It sort of reminds me of the Helmet law wars the bikers used to have with the Government. For every proof (with studies) of the safety of helmets there was  also a counter (with studies) of the danger.  It seems the same with diet.  I feel like I'm watching a tennis match...lol. My head goes back and forth, back and forth...

  • rhc

    10/15/2010 1:43:22 PM |

    @Tommy
    I totally agree! I've been on both sides and they both have their 'scientific' proof. Also, everyone seems to want to or have to lose weight. That too is quite frustrating for me since I've been slender all my life and have no high BP or triglycerides - just have to watch my blood sugar. And here too both sides have their proof that it works. UGHHH!!!

  • PJNOIR

    10/15/2010 2:15:12 PM |

    I've tried these and really have tried to give them a place but they taste like rubber. The worst.

  • Carl

    10/15/2010 2:46:02 PM |

    Spaghetti squash. Problem solved.

  • Derek H

    10/15/2010 7:58:13 PM |

    Right on Carl, spaghetti squash rocks.

  • Eva

    10/16/2010 4:54:32 AM |

    SHiritaki noodles with soy are typically about 20% soy so that's not going to be a ton of soy unless you eat them often.  The other 80% is fiber from a tuber.  You actually don't need to boil these noodles, just rinse well and then add to your dish at the last minute to heat them.  Over cooking makes them more rubbery.  YOu really only need to heat them.  They also will NOT soak up liquid so make sure your sauce is plenty thick before adding the noodles.  If anything the noodles tend to release a bit of water back into the dish.  I don't normally eat soy but am OK with the small amount in an occasional dish of shiritaki noodles.

  • Alex

    10/17/2010 11:37:06 AM |

    I'm very sensitive to starches, and grains, pseudo-grains, and starchy tubers all spike and crash my blood sugar. Beans, however, do not. When I want to indulge in pasta, I buy mung bean fettuccine at the healthfood store. They're made from whole mung beans, not refined mung bean starch, like the translucent, mung bean based, Asian noodles.

  • Anonymous

    10/18/2010 8:38:20 PM |

    I use zucchini and yellow squash as a great low-carb replacement for noodles. Not only do they lack carbs, but they are a decent source of some vitamins and minerals.

  • carpjm

    10/26/2010 5:21:10 AM |

    Check out miraclenoodle.com, they have the soy free and have tons of varieties, try the orzo!!!

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